The emergence of variants of concern in late 2020 marked a shift in the COVID-19 pandemic.
The Delta variant: What it is, where it came from, why it’s different and whether COVID-19 vaccines can prevent it.
The SARS-CoV-2 virus is mutating.
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The virus is evolving and new strains are more transmissible. Will the vaccines work against these new variants? How can researchers stay ahead of the virus’s evolution?
I am a physician, and I just got my first shot of the Pfizer COVID-19 vaccine.
With vaccines forthcoming for most Americans, many groups, including expectant mothers, are wondering if the vaccine is safe for them and their babies. A physician-scientist explains.
The French government will not accept any passengers arriving from the U.K. amid fears over the new mutant coronavirus strain.
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A new strain of the SARS-CoV-2 virus that causes COVID-19 appears to be spreading fast in the UK. This probably isn’t a big problem, but the data isn’t in yet.
This antibody adopts a Y-shape. The arms of the Y make up the part of the antibody that binds to the target.
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Antibodies are great for neutralizing viruses. But they are big and bulky. Antibody engineers are now creating smaller synthetic antibody-like molecules that may be better for fighting COVID-19.
Although they remain untested as a treatment, this suggests these drugs could be used as a therapeutic against the coronavirus.
The spike protein on SARS-CoV-2 interferes with pain perception.
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The SARS-CoV-2 virus usually infects the body via the ACE2 protein. But there is another entry point that allows the virus to infect the nervous system and block pain perception.
Experts are confident that there will be a vaccine next year.
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As grim as things are with the pandemic raging in the US and the mounting death toll, there are many reasons to be optimistic there will be a vaccine by early next year.
We are slowly figuring out which drugs and therapies are effective against the new coronavirus.
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During the last six months, news reports have mentioned dozens of drugs that may be effective against the new coronavirus. Here we lay out the evidence and reveal which ones are proven to work. Or not.
The good news is: you’ll probably get it back.
Severe COVID-19 may leave lasting scars in the lungs, but some recovery could happen over time.
Researchers have been trying to understand a possible link between blood pressure medications and COVID-19.
The use of antihypertension medication during the coronavirus pandemic has been a subject of hot debate but people should be cautious about simple conclusions.
Here’s what we know about immunity to COVID-19, and what that means for getting life back to normal.
A molecular model of the spike proteins (red) of SARS-CoV-2 binding to the angiotensin-converting enzyme 2 (ACE2) protein, the receptor (blue) which is its the entry route to the target cell.
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The ACE2 receptor allows the virus that causes COVID-19 to infect and destroy our cells. What is the normal role of ACE2 in the body, and could it be the key to blocking infection?
A molecule responsible for lowering our blood pressure also helps coronavirus get into our cells and replicate. And it occurs more in men than in women.
What would Darwin consider the best adaptation to protect against the coronavirus?
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Who is most likely to survive an infection of the new coronavirus? Two immunologists explain that it is those who mount exactly the right immune response – not too weak, not too strong.
People with heart conditions at higher risk of severe COVID-19. But coronavirus appears to affect the heart directly, too.
A woman with diabetes monitors her glycemia on the eighth day of a strict lockdown in France aimed at curbing the spread of COVID-19.
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What does high blood sugar have to do with vulnerability to COVID-19? And is there a role for the controversial drug hydroxychloroquine in lowering blood sugar in COVID-19 patients?
A patient is brought into the emergency room of the Wyckoff Heights Medical Center in Brooklyn, New York.
Some things about COVID-19 still don’t add up.