Transmission electron micrograph of particles of the SARS-CoV-2 virus.
The SARS-CoV-2 virus at the origin of the Covid-19 pandemic is one ten-thousandth of a millimeter in diameter. How can such a microscopic organism have such an immense impact on global health?
Access to testing had been improving across the U.S., but as cases increase, more testing is needed.
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COVID-19 cases are skyrocketing across the US. Testing has ramped up over the past few months, but increasing hospitalizations, deaths and test-positivity rates show that the virus is out of control.
Estimating the cost of antibiotic resistance to economies and health-care systems is fraught with difficulty, but new research says Australia will be hit harder than we think.
Y-shaped proteins called antibodies are vital for attacking and destroying the virus.
Monoclonal antibodies are synthetic molecules manufactured in the lab. But do we need them if a vaccine is on its way?
A man in San Pablo, California, gets a flu shot at a drive-through flu shot clinic Nov. 6, 2014.
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Many people object to the added ingredients in vaccines. But pharmacists explain why those fears are unwarranted.
Holiday events will need to be a little different due to the pandemic.
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COVID-19 and holiday family gatherings are not a good pair. But taking the right precautions before, during and after the family gets together can greatly reduce coronavirus risk this holiday season.
In patients with MS, the body’s immune system attacks the nerve cells.
This antibody protects patients from viruses, even while on immunosuppressant drugs.
Under relaxed public health restrictions, deaths will spike far before herd immunity is achieved.
AP Photo/Mark Lennihan
Some have suggested the US allow healthy people to return to normal life, catch the coronavirus and get the population to herd immunity. The science says this plan is doomed to fail from the start.
In autoimmune diseases, circulating antibodies destroy an individual’s own tissues.
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Are antibodies that attack a patient's own organs contributing to severe forms of COVID-19? A new study suggests specific antibody tests that may reveal the answer.
The Spike protein on the surface of SARS-CoV-2 must bind to proteins on the surface of human cells to trigger an infection.
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Scientists in the UK and Germany discovered a new doorway that the COVID-19 virus uses to infect human cells. This reveals new therapeutic possibilities for blocking the virus.
Using music to represent the coronavirus sequence gives us a new way to think about and understand the genome.
Vaccines work by teaching your immune system about new viruses. Your immune cells are very clever – they will remember what they learnt, and protect you if you encounter that virus in the future.
Are patients with severe COVID-19 victims of their own immune response?
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Patients suffering from severe COVID-19 may be experiencing a rogue antibody response similar to that seen in autoimmune diseases. The findings offer new approaches for COVID-19 therapy.
Hepatitis C led to an estimated 400,000 deaths in 2016.
Michael Houghton, an Edmonton-based virologist, was one of the recipients of this year's Nobel Prize for physiology or medicine for the discovery of hepatitis C.
Classroom experiments show how the coronavirus can spread and who’s at greatest risk.
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Experiments in college classrooms show how tiny respiratory droplets known as aerosols can spread, even with good ventilation. The risk isn't the same in every seat.
The microbes in the mother’s gut can alter the number of neurons in the baby’s brain and the connections they make.
Microbes in the gut aren't just important for digesting your food. In pregnant women, these gut microbes are producing chemicals that are essential for proper brain development of the fetus.
When a person sneezes, tiny droplets, or aerosols, can linger in the air.
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Scientists explain what you need to know about the COVID-19 risks in the air, from how aerosols form to how to keep kids safe on a school bus.
The spike protein on SARS-CoV-2 interferes with pain perception.
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The SARS-CoV-2 virus usually infects the body via the ACE2 protein. But there is another entry point that allows the virus to infect the nervous system and block pain perception.
Death rates vary by demographic, with age and race playing big roles.
AP Photo/Kathy Willens
Using random testing, researchers in Indiana were able to calculate death rates by age, race, and sex and found sharp increases in risk of death among older and non-white state residents.
Children run as an agent of the National Institute of Public Hygiene carries out fumigation in the Anyama district of Abidjan,Ivory Coast.
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A warming climate may change the types of viruses that thrive. A new report suggests that the threat of malaria may be replaced by dengue, for which there is no treatment and no cure.